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Ongoing irritation in eosinophilic esophagitis (EoE) results in fibrostenosis, leading to dysphagia. Endoscopy stays the criterion customary for diagnosing EoE and monitoring remedy effectiveness. Nonetheless, the present definition of remission for EoE depends totally on counting eosinophils per high-power microscopic subject (hpf). To establish persistent molecular drivers of the illness, Hoelz et al (p 505) investigated molecular alterations in pediatric sufferers with EoE in histologic remission utilizing tissue transcriptomics and proteomics.

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