Nonsteroidal anti-inflammatory medication (NSAIDs) are among the many most consumed medicines worldwide and the primary drug hypersensitivity triggers. The most typical sort is bred by pharmacological cyclooxygenase (COX)-1 inhibition, after chemically-unrelated NSAID consumption, and NSAID-induced acute urticaria/angioedema (NIUA) is essentially the most frequent scientific phenotype, with sufferers being regularly sensitized (atopic) to Dermatophagoides pteronyssinus (DP).
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