Bronchial bronchial asthma (BA) is a standard power illness affecting 300 million individuals globally,1 with as much as 10% of sufferers presenting with extreme BA. BA is a fancy respiratory illness characterised by airway irritation, bronchial hyperresponsiveness, and variable airflow limitation2 and is usually difficult by rhinitis and power rhinosinusitis with nasal polyps (CRSwNP).3 BA is heterogenous, with variable phenotypes and endotypes, and subsequently could be tough to diagnose and deal with.4 Whereas the one-airway–one-disease speculation proposed that the higher and decrease airways share the same pathophysiology,5 the mechanisms underlying BA are various, with completely different and typically overlapping endotypes.
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